Ebola Hellfire


Fair Use:  Caption: Colorized scanning electron micrograph of filamentous Ebola virus particles (blue) budding from a chronically infected VERO E6 cell (yellow-green).
Credit: National Institute of Allergy and Infectious Diseases, NIH

Biological Convergence for Onslaught

The Ebola biological wildcard is dealt, whether real or perceived, and we all must bear the consequences of government and researcher’s abhorrent mischief.   This is an orchestrated effort by dark powers and principalities to depopulate the world and further wither the spirit of humanity at a time of great spiritual iniquity.   The blood moon is rising for a second time within a year and it is the season for blood sacrifice. Continue reading

Biological Onslaught Against Humanity is Unleashed

  Image from government CBRNe website

By Celeste

Victory over humanity has begun in earnest by assaulting the body with Chemical, Biological, Nuclear, Radiological and e-tools which are the wild card.  E-tools are interchangeable and can be explosive, environmental, economic, or other. CBRNe is applied in biblical proportion to overwhelm the human body to the degree that some will become disabled and others succumb to death.  Additional stresses such as spiritual oppression and physical deprivation (War on Calories) will diminish cognition, the ability to accomplish menial tasks, and compound the death rates.       Continue reading

Natures First Aide Kit: Desert Parsley: Influenza


Lomatium dissectum

 Desert Parsley

  For educational and informational purposes only. 

Not for diagnosing, treating, curing, mitigating or preventing any disease.  Seek advice from a licensed, qualified health-care professional.


Desert Parsley is one of two wild plants that you do not want to be without in this age of viral infections.  Despite it’s name it grows in a wide variety of regions where it is dry, typically preferring rock slide areas. 

Desert Parsley is in the carrot family and older plants have very large rutabaga-potato looking tuber-roots.  Roots can weigh up to 15 pounds in older plants.  This wild plant is adaptogenic which means it will heal the part of your body that needs healing without you doing a thing!  Typically a person would harvest 4-5 pounds per person for influenza season but we have found that it is so potent very little is really needed to stop the flu dead in its tracks.  Continue reading

Pandemic Woes: Weather Influences Influenza and Pneumonia

No duh…they already knew this but for those who do not study history it is new to them.

From 2009:  Pandemic Woes:  Bad Weather Ahead? (1918 & 2009)

The Impact of Weather on Influenza and Pneumonia Mortality in New York City, 1975-2002:

A Retrospective Study

The substantial winter influenza peak in temperate climates has lead to the hypothesis that cold and/or dry air is a causal factor in influenza variability. We examined the relationship between cold and/or dry air and daily influenza and pneumonia mortality in the cold season in the New York metropolitan area from 1975-2002. We conducted a retrospective study relating daily pneumonia and influenza mortality for New York City and surroundings from 1975-2002 to daily air temperature, dew point temperature (a measure of atmospheric humidity), and daily air mass type. We identified high mortality days and periods and employed temporal smoothers and lags to account for the latency period and the time between infection and death. Unpaired t-tests were used to compare high mortality events to non-events and nonparametric bootstrapped regression analysis was used to examine the characteristics of longer mortality episodes. We found a statistically significant (0.003) association between periods of low dew point temperature and above normal pneumonia and influenza mortality 17 days later. The duration (0.61) and severity (0.56) of high mortality episodes was inversely correlated with morning dew point temperature prior to and during the episodes. Weeks in which moist polar air masses were common (air masses characterized by low dew point temperatures) were likewise followed by above normal mortality 17 days later (0.019). This research supports the contention that cold, dry air may be related to influenza mortality and suggests that warning systems could provide enough lead time to be effective in mitigating the effects.


Robert E. Davis1*, Colleen E. Rossier1, Kyle B. Enfield2

1 Department of Environmental Sciences, University of Virginia, Charlottesville, Virginia, United States of America, 2 Division of Pulmonary and Critical Care, Department of Medicine, University of Virginia Health System, Charlottesville, Virginia, United States of America


Editor:  Viboud, National Institutes of Health, United States of America

In Progress: One Health Davos Summit Yesterday

Introducing One Health – One Planet – One Future: International Perspectives

Yesterday marked the opening of the first GRF One Health Summit in Davos, Switzerland. The One Health Summit is being held at the renowned international conference center in Davos from 19-23 February 2012. Two hundred and seventy (270) delegates are present from more than 60 countries. The conference is co-hosted with the Swiss Tropical and Public Health Institute. Basel, and the Swiss Institute of Allergy and Asthma Research, Davos, with help from 38 collaborating institutions. At the conference, there are eight plenary and 15 parallel sessions, comprising 125 papers and 16 poster presentations.

Alain Vandersmissen described the long, historical relationship between human health and animal diseases, starting with developments in ancient Babylonia. The question first began to be tackled seriously in the 19th century, but it was not until 1976 that the concept of One Medicine emerged. This was gradually broadened and transformed into the One Health concept, which in 2004 produced the 12 Manhattan Principles. By 2007, promoting One Health became a natural extension to the global response to avian influenza. A strategic framework evolved in October 2008 and the first One Health conference was held in Canada in 2009. By the Hanoi conference of 2010 the emphasis on avian influenza had broadened to embrace animal influenza as a whole. By the Melbourne conference of 2011 the scientific world had been joined by representatives of the private sector and civil society. Hence it has broadened from an approach to a movement. Continue reading

Pandemic Woes: 1918 Influenza Pandemic: US Army Perspective

Bird Flu… Swine Flu… 1918 Flu.  Just what does this flu do to merit the massive attention that media and government are seizing upon?  There is not allot written about the 1918 influenza because there was a media black-out due to global events.  The following is a vivid written and photographic description of the 1918 influenza from the US Army perspective.

From the trenches….


Accessed Under Fair Use from Suburban Emergency Management Project

(now purged from the internet)

Biot #641: August 16, 2009

In the fog of the influenza pandemic of 1918, Dr. Victor C. Vaughan, head of communicable diseases for the U.S. Army training camps, cautioned shaken observers that a full accounting of the pandemic required time and further study. (1) Surgeon General M.W. Ireland and Lieutenant Colonel Joseph F. Siler, M.D., prepared and delivered that final accounting in their 600-page tome titled The Medical Department of the United States Army in the World War: Communicable and other Diseases (1928). (2-4) Major Milton W. Hall, M.D., prepared the 110-page chapter on the respiratory disease outbreaks.

Surgeon General Merritte Weber Ireland. Source: http://upload.wikimedia.org/wikipedia/en/e/e5/Merritte_Weber_Ireland.jpg; accessed August 2, 2009.

U.S. Army camp hospital administration building, World War I, where record keeping likely was performed. Source: http://history.amedd.army.mil/booksdocs/wwi/MilitaryHospitalsintheUS/chapter4figure15.jpg; accessed August 2, 2009.

This work on the influenza pandemic and other disease outbreaks among American soldiers between April 1, 1917 and December 31, 1919 is based on massive amounts of actual data analyzed and interpreted by clinicians, medical epidemiologists, and scientists for use by posterity (us). The book is the most detailed, comprehensive, complete, reliable, valid and useful description of the 1918 influenza pandemic available today, in this author’s opinion. Its scientific approach contrasts with the social historical approach common to many books available today on the 1918 influenza pandemic.

What did the physician authors of The Medical Department of the United States Army in the World War: Communicable and other Diseases learn from the medical data collected during the Great War? What can people living a century later (us) learn from their work?

1. Many Infectious Diseases Flourished before and during the Influenza Pandemic Era, 1917-1919, but Influenza and its Complications Dominated

During World War I in the U.S. Army camps, many diseases flourished. There are individual chapters in The Medical Department of the United States Army in the World War: Communicable and other Diseases on the typhoid and the paratyphoid fevers, inflammatory diseases of the respiratory tract, tuberculosis, cerebrospinal meningitis, anthrax, diphtheria, the venereal diseases, the diarrheal group of diseases, chickenpox, scarlet fever, measles, mumps, German measles, encephalitis lethargica, infectious jaundice, typhus fever, trench fever, Vincent’s disease, the malarial fevers, intestinal parasites, diseases of the skin, and neurocirculatory asthenia. Of these myriad diseases afflicting soldiers in the U.S. Army during World War I, the respiratory diseases dominated in quantity and severity. (4) Dr. Hall declared, “[T]he serious and fatal inflammations of the respiratory tract…formed by far the most important factor in the sickness and death records of the Army during the World War.” (5)

Receiving building at a base hospital in a U.S. Army camp, World War I. This is not the hospital ward itself. Instead it is where sick soldiers went for evaluation and triage. Source: http://history.amedd.army.mil/booksdocs/wwi/MilitaryHospitalsintheUS/chapter4figure16.jpg; accessed August 2, 2009.

Interior of one-story hospital ward, U.S. Army camp, World War I. Source: http://history.amedd.army.mil/booksdocs/wwi/MilitaryHospitalsintheUS/chapter4figure26.jpg; accessed August 2, 2009.
2. Morbidity and Mortality Data, Influenza and other Contagious Diseases, U.S. Army, Great War

The total mean strength of the U.S. Army (officers and enlisted men, henceforth, soldiers) from April 1, 1917, to December 31, 1919, was 4,128,479. (6-7) Of these 4,128,479 soldiers, 3,515,464 (85%) reported sick for admission. [!] (6) This number of sick soldiers is astounding.

The total number of soldiers admitted for respiratory diseases was 1,125,401, which calculates to 27% of all soldiers in the U.S. Army and 32% of U.S. soldiers reporting sick for admission. In other words, one out of three soldiers developed respiratory disease requiring sick admission at some time between 1917-1919 and one out of three soldiers reporting for sick admission carried a respiratory disease diagnosis. Note that the respiratory disease diagnoses in the respiratory disease category employed by Dr. Hall were limited to influenza, bronchitis, broncho-pneumonia and lobar pneumonia. Each of these disease entities affects the lower respiratory tract. Upper respiratory disease categories such as pharyngitis [sore throat], tonsillitis, and sinusitis were not included in Dr. Hall’s respiratory diseases category.

Of the 1,125,401 soldiers admitted for respiratory disease diagnoses, 791,907 carried the diagnosis of influenza, which calculates to 19% of all soldiers in the U.S. Army (1917-1919) and 23% of all soldiers who reported sick for admission (1917-1919). By contrast and by way of example, 67,026 (2%) soldiers reported sick for syphilis, 4,831 (0.1%) for cerebro-spinal meningitis, and 1,529 for typhoid fever (0.04%). (2)

Of the 791,907 soldiers diagnosed with influenza, 24,664 (3%) died. (8) There were so many deaths, logistical problems arose over managing the bodies, according to a congressional probe to which Secretary of War Newton Baker responded in three days of testimony in late January 25, 1918, available elsewhere. (9) The U.S. Congress was having difficulty obtaining information on U.S. Army camp operations because of censorship of the press by the Wilson administration, which did not want Germany to know, via U.S. media, of troubles affecting the raising of a U.S. army. Of the 791,907 soldiers diagnosed with influenza, 767,243 (97%) survived their illness.

Of all the deaths charged to influenza, 99.4% were recorded as due secondarily to pneumonia (66.1% broncho-pneumonia and 33.3% lobar pneumonia; the difference between the two types of pneumonias is not important here; rather the fact that the patients died a respiratory death is what is important).

The total number of deaths from all diseases during the war was 58,119. Of the 58,119 deaths due to all diseases, the respiratory diseases accounted for 46,992, or 80.85%. (8) In other words, four out of five soldiers who died of disease died of respiratory disease (as defined by Dr. Hall, see above).
3. Problems Defining a Case of Influenza during the Great War

At the beginning of 1918, most clinicians believed that the bacillus of Pfeiffer caused influenza. Dr. Richard Pfeiffer was an eminent German researcher and son-in-law of Dr. Robert Koch whose conclusions that bacteria caused influenza were based on work done late in the 1889-1892 cycle of the disease. However, noted Dr. Hall, “With the advent of the earlier recognizable waves of the 1918 outbreak it became evident that the bacillus of Pfeiffer was not uniformly present in the cases examined.” (10) Bacteriologists simply were not growing the Pfeiffer bacillus (today known as Haemophilis influenza) from sputum, blood, or other fluids of the gravely sick soldiers exhibiting signs of what was being called “influenza.” In fact, instead, bacteriologists were growing pneumococci and other “mouth” bacteria from sputum, which were under normal circumstances considered benign.

Pfeiffer’s bacillus (Haemophilus influenza) under the microscope. Source: http://pathmicro.med.sc.edu/Infectious%20Disease/Hinfluenzae1.jpg; accessed August 2, 2009. Continue reading

Pandemic Woes: Bad Weather Ahead?

The Island Queen in distance being pushed down the Ohio River by the ice. Source: http://www.cincinnativiews.net/images/Flood%20&%20Ice%20Gorge%2017rp.jpg; accessed July 23, 2009.

Fair Use Access Suburban Emergency Management Project,July 5, 2009

(now purged from the internet)

From the trenches,


Biot #637: July 23, 2009

Meteorologist Preston C. Day (1859-1931) wrote in December 1918, “The severity of the weather experienced during December and January of the winter of 1917-1918 over the greater part of the United States east of the Rocky Mountains, and also over much of Canada and Alaska during the early part of the period, was so unusual as to the length of time the low temperatures persisted, the great area involved, and the degree of cold maintained, that some discussion of the contributing factors, and comparison with similar occurrences of previous years, seems desirable.” (1-2)


The sinking of the Princess in the Ohio River, winter of 1917-1918. Source: http://www.cincinnativiews.net/images/Flood%20&%20Ice%20Gorge%2018rp.jpg Continue reading

Sinusitis Relationship to Influenza

Influenza Sinusitis and its Relation to Epidemic Influenza

Free Access from Suburban Emergency Management Project August 4, 2009, Biot #638

(This has been purged from the internet)

July 27, 2009

H.E. Robertson, M.D. of Minneapolis, Minnesota, became a major in the U.S. Army in 1917, serving overseas in the Central Medical Department Laboratory of the American Expeditionary Force in France. In May 1918, he published an article titled “Influenza-sinus disease and its relation to epidemic influenza” in the Journal of the American Medical Association, based on his observations of patients in the hospital of the British Expeditionary Forces in France. May 1918 was during the first wave of pandemic influenza (see graph below). His observations are astute.

Graph showing three pandemic waves: weekly combined influenza and pneumonia mortality, United Kingdom, 1918-1919. Source: http://www.cdc.gov/ncidod/eid/vol12no01/05-0979-G1.htm; accessed July 27, 2009.

Robertson wrote, “A typical attack of influenza usually beings as a rhinitis and spreads from the nasal mucosa throughout the respiratory tract. As the chief symptoms are produced by the involvement of the bronchial tree, attention is naturally directed to the lungs and bronchi as the most important foci for the infection. However, in studying acute cases one is often impressed by the fact that the severity of the disease, best evidenced by the general prostration of the patients, is out of all proportion to the physical signs. Tracheo-bronchitis with abundant muco-purulent sputum, a particularly distressing and often spasmodic cough, and moist rales [wet sounds] uniformly present in both lungs, almost exhaust the positive findings of the respiratory tract. It becomes difficult to understand why a vigorous, previously healthy young adult, should succumb to an infection which in many cases appears to be almost wholly confined to the tracheo-bronchial mucosa. It might even be questioned whether such an individual should die from a primary bronchitis and broncho-pneumonia. In the opinion of many clinicians and pathologists, such a combination is rather rare. (1)

Robertson believed that “in addition to the infection of the respiratory tract per se, “other lesions, almost uniformly neglected by writers on the subject and rarely noted by clinicians or pathologists, may prove to be very seriously important factors both in the clinical picture and in the often fatal outcome of the disease.” (1) What are these lesions?

In October 1917, Robertson says he visited the base hospital zone of the British Expeditionary Forces in France, at which time “Captain Rolland of the Royal Army Medical Corps mentioned his observations in a series of cases of this disease, which had occurred in epidemic proportions and which was highly fatal. The influenza bacillus seemed without doubt to be the causal agent, though pneumococci and streptococci were often also isolated [in the laboratory].” Robertson, however, was most surprised at the “involvement of the sinuses at the base of the skull” during autopsies of patients who had succumbed to influenza.

Robertson noted, “Infection of the accessory sinuses of the nose and skull have [sic] often been noted. As early as 1837, Petrequin stated that frontal sinus involvement produced the headache so often present in [influenza]. Bronchin, in 1884, distinguished the ‘cephalic’ form of influenza by localizations in the nasal fossae, and the frontal and maxillary sinuses. Pfeiffer, however, in his classic description of the etiology and pathology of influenza, does not mention the sinuses. [2]…[Pfeiffer] evidently did not encounter or missed the significance of any sinus complications,” declared Robertson. “In short, sinus disease in epidemics of influenza is either totally disregarded or described as an accidental complication or sequel of the infection of the pulmonary tract.” Robertson believed otherwise.

Location of human sinuses. Source: http://ent.med.nyu.edu/files/ent/u3/nose_anatomy_front.gif; accessed July 27, 2009.

“When, therefore, our first fatal case of influenza tracheo-bronchitis and broncho-pneumonia showed, at postmortem examination, an empyema [collection of pus] of both sphenoid [sinus] cavities, and the pus from these revealed both smears and cultures typical influenza bacilli, the condition was regarded as an interesting but unusual complication. However, as case after case coming to necropsy showed similar or comparable lesions, the circumstances warranted more careful and detailed study,” Robertson noted.

Robertson summarizes fourteen necropsies showing involvement of the sinuses.

In his comment section, he wrote, “It would, of course, be unreasonable to assert that all cases of influenza are accompanied by sinus complications…The number of cases is altogether too small to justify any sweeping conclusions. They do serve most emphatically to emphasize the importance of the sinuses in the respiratory type of influenza.” Why?

First, infection of the sinuses “constantly menaces the pulmonary system, only awaiting suitable conditions of exposure and lowered resistance for hostile invasion, but also furnishes continued sources of toxic absorption, not to mention the direct effect on the well being of the patient from the presence of these local conditions.”

Second, and more important, “is the bearing that these local infections have on prophylaxis and treatment. When their attention had been called to the possible constant presence of sinus disease in patients suffering from influenza bronchitis, the attending physicians adopted local measures of treatment for these conditions, even when their presence could not be diagnosed with any degree of certainty. Local applications to the nasal passage of cocaine and epinephrine solutions often resulted in copious discharges of thick, muco-purulent exudates from the sinuses, with marked relief to the patient, such as amelioration of headache and pain in the eyes, as well as definite betterment of the conditions in the bronchi and trachea.” (1)

Detail of human sinuses. Source: http://www.sinustreatmentcenter.com/scfig3_500.jpg; accessed July 27, 2009.

Robertson summarizes his findings, as follows (two have been omitted):

1. “Epidemics of respiratory influenza (purulent tracheo-bronchitis) have been fairly severe in both the American and the British Expeditionary Forces.
2. In the investigation of cases, both clinically and at postmortem, little attention in the past has been give to the question of accompanying sinus disease.
3. Of eight fatal cases of purulent tracheo-bronchitis due to the influenza bacillus, all but one showed involvement of one or more of the sinuses at the base of the skull by inflammatory processes, probably, in all cases, directly due to the invasion of these sinuses by the influenza bacillus.
4. Appropriate treatment of the sinuses in patients suffering from influenza often served to relieve the symptoms and apparently to hasten convalescence.
5. Investigation of the sinuses during epidemics of influenza is strongly recommended and urged not only on therapeutic but also on prophylactic grounds.” (1)

Robertson’s findings are relevant to today. Effective treatment of influenza sinusitis may prevent spread to the lungs. Treatment, however, is possible only with awareness of the disease entity, i.e., influenza sinusitis.


1. H.E. Robertson: “Influenzal sinus disease and its relation to epidemic influenza.” JAMA, May 1918, Volume 70, Number 21, pp. 1533-1535. Available at
2. Pfeiffer’s bacillus, subsequently named Hemophilus influenza, was shown by Martha Wollstein, M.D., in 1919, to be “at least a very common secondary invader in influenza, and that its presence influences the pathological process.” However, patients’ serological reactions that she studied were not “sufficiently stable and clean-cut to signify that Pfeiffer’s bacillus is the specific inciting agent of epidemic influenza.” Source: Martha Wollstein: “Pfeiffer’s bacillus and influenza: A serological study.” The Journal of Experimental Medicine, Volume 30, pp. 555-568, 1919. Abstract available at http://jem.rupress.org/cgi/content/abstract/30/6/555; full text is available at http://jem.rupress.org/cgi/reprint/30/6/555; accessed July 27, 2009.

Copyright 2009 – SEMP INC., All Rights Reserved.

From the trenches,


Pandemic Woes: Who Does What? A Simple Org Chart

Who are the lead agencies for what during a flu event?  We also are under the UN direction since the declared Level 6 .

Don’t forget the get your Freedom Packs Ready and No Constitutional No Trespassing Signs up and ready for ‘visitors’.  Have some way to copy or reproduce individual identification to accompany the Public Servant Questionnaire.


Priority # Priority Title Lead Department

1 Mass Vaccination    HHS/CDC
2 Public Health Continuity of Operation Plan   HHS/CDC
3 Surveillance and Laboratory   HHS/CDC
4 Communication   HS/CDC
5 Community-Wide Healthcare Coalitions to meet Patient Surge Expected from Pandemic Influenza     HHS/ASPR/HRSA
6 Facilitating Medical Surge  HHS/ASPR/HRSA
7 Fatality Management  HHS/ASPR/HRSA
8 Antiviral Drug Distribution Plan – submitted separately  HHS/CDC
9 Community Containment Plan – submitted separately   HHS/CDC
10 Policy Process for School Closure and the Communication Plan for this Decision ED
11 Education and Social Services in the Face of School Closures  ED
12 Sustain/Support 17 Critical Infrastructure Sectors & Key Assets DHS
13 Working with the Private Sector to Ensure Continuity of Operations for Critical Essential Services so that Critical Infrastructure Operations are as “Near Normal” as Possible for Social and Economic Well-Being DHS
14 State Plans Must Conform to all NRP/NIMS Requirements   DHS
15 Mitigate the Impact of an Influenza Pandemic on Workers in the State DHS
16 Assisting Employers in the State   DHS
17 Employment Policies during an Influenza Pandemic  DHS
18 Human Resource Policies for State Employees DHS
19 Coordination of Law Enforcement  DOJ
20 Critical Essential Function for Food Safety  USDA, HHS/FDA
21 Operational Status of State-Inspected Slaughter and food Processing Establishments Including Talmadge Aiken Plants  USDA, HHS/FDA
22 Communication Strategy for Interface with USDA food Safety Inspection Service and FDA’s Federal State Relations   USDA, HHS/FDA
23 Ensure Adequate Reporting Systems Regarding Food Safety USDA, HHS/FDA
24 State Advisories Regarding Diplomatic Missions  DOS

From the trenches,


Swine Flu (H1N1) Economic Update

Potential economic impacts of the A H1N1flu outbreak

Fair Use Access June 23, 2009 World Bank June 22, 2009 GDF Report

For Educational Purposes Only

Although the spread of H1N1 appears to have eased, its spread is likely to pick up as the flu season begins in the southern hemisphere and again when it returns in the northern hemisphere. Even if it does not mutate into a more deadly form, a second wave of the flu in low-income countries’ could have serious consequences-given poor countries limited capacity to monitor and treat an outbreak and the higher incidence of chronic disease within their populations (the  re-existence of chronic health conditions and delays before medical intervention appear to be among the factors that have contributed to deaths where they have occurred). More worrisome is the possibility that H1N1 could mutate into or combine with a more aggressive form of the flu-such as H5N1 (avian influenza). As a flu for which much of the world’s population has limited pre-existing immunity (WHO 2009), A H1N1 could infect as much as 35 percent of the world’s population (WHO 2006)-spreading throughout the world in as few as 180 days during flu season. Continue reading